Hypoxemia in Man Induced by Prolonged +Gz Acceleration

Item

Title
Hypoxemia in Man Induced by Prolonged +Gz Acceleration
Date
1963
Index Abstract
Not Available
Photo Quality
Not Needed
Report Number
AMRL TDR 62-137
Creator
Bjurstedt, H.
Corporate Author
Laboratories of Aviation and Naval Medicine, Karolinska Institutet, Stockholm, Sweden
Laboratory
Biomedical Laboratory
Extent
15
Identifier
AD0411465
Access Rights
OTS
Distribution Classification
1
Contract
AF 61(052)-153
DoD Project
7220
DTIC Record Exists
No
Distribution Change Authority Correspondence
None
Distribution Conflict
No
Cover Price
0.5
Abstract
Changes in the arterial oxygen saturation were re corded in healthy subjects on the human centrifuge by continuous cuvette oximetry before, during and after prolonged exposures to positive accelera tion. With the subjects breathing air and wear ing an automatically inflated anti-G suit, an im mediate fall in the arterial O2 saturation was observed upon exposure to 4.5-5.0 Gz. After one minute of the first exposure the O2 satura tion ranged between 95 and 81 per cent, the arterial pH remaining essentially unchanged. At the same time the respiratory minute volume had increased, indicating gross deterioration in the efficiency of pulmonary function. Re peated exposures caused the arterial O2 satura tion to fall at a faster rate and to a lower level with each consecutive run. The rate of resatura tion on returning to normal gravity was usually slow, and markedly so after several exposures. The last-mentioned observations are interpreted as being mainly the result of residual atelec tasis in dependent regions of the lungs. The potential dangers of acceleration-induced hypoxemia in high performance flight missions are discussed.
Report Availability
Full text available
Date Issued
1963-04
Provenance
RAF Centre of Aviation Medicine
Type
report
Format
1 online resource
Media
AMRLTDR62-137.pdf